I found mentions of this and other arylalkylaminoproprionamide LSD analogs in the abstracts,
Mol constitution was related to inhibitory activity assayed with isolated human plasma pseudocholinesterase. The inhibitors of this series were less potent than lysergic acid diethylamide, but retained sufficient activity to illustrate relations between chem structure and biochemical response.
how does LSD interfere with cholinesterase in humans? (as the abstract so implies.) and how does cholinesterase inhibition relate to the psychoactivity of the various LSD derivitive?